An amyloid β42-dependent deficit in anandamide mobilization is associated with cognitive dysfunction in Alzheimer’s disease.

“The endocannabinoids and their attending cannabinoid (CB)(1) receptors have been implicated in the control of cognition, but their possible roles in dementias are still unclear.

 The results suggest that an Aβ(42)-dependent impairment in brain anandamide mobilization contributes to cognitive dysfunction in AD.”

http://www.ncbi.nlm.nih.gov/pubmed/21546126

One thought on “An amyloid β42-dependent deficit in anandamide mobilization is associated with cognitive dysfunction in Alzheimer’s disease.

  1. I do consider all the ideas you have offered on your post. They’re really convincing and will certainly work. Still, the posts are very quick for starters. May you please prolong them a bit from subsequent time? Thanks for the post.

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