Juicing cannabis miraculously saves lives after physicians declare the battle lost

“(NaturalNews) At 16, Kristen Peskuski was suffering from joint inflammation and an array of autoimmune conditions which made her organs and other tissues swell, including interstitial cystitis and lupus. She was prescribed over 40 different anti-inflammatory, antibiotic and painkilling medications to combat the symptoms. Still struggling to bring the symptoms under control, Kristen developed steroid toxicity. She was told that the most she could hope for was reduced discomfort, and with luck, she might make it to her 30th birthday. Seeking alternative treatments, she began juicing raw cannabis leaves every day, and within two months, Kristen’s back pain had been eliminated, and she had stopped using any other painkillers.”

Learn more: http://www.naturalnews.com/035400_juicing_cannabis_remedies.html#ixzz29OcuQ7gQ

American Cancer Society on Cannabis curing Cancer

“scientists reported that THC and other cannabinoids such as CBD (cannabidiol) slows growth and/or causes death of certain types of cancer cells growing in laboratory dishes. Some animal studies suggest certain cannabinoids may slow growth and reduce spread of some forms of cancer”

http://www.cancer.org/Treatment/TreatmentsandSideEffects/
ComplementaryandAlternativeMedicine/HerbsVitaminsandMinerals/marijuana

Cannabinoid receptor activation correlates with the pro-apoptotic action of the β2-adrenergic agonist, (R,R’)-4-methoxy-1-naphthylfenoterol, in HepG2 hepatocarcinoma cells

“In the last several years, development of new CBR ligands has become an intense area in cancer research because of the role of the endocannabinoid system in the regulation of cell proliferation and apoptosis…

The therapeutic potential for synthetic small bivalent ligands holds great promise as new lead compounds in a wide range of disparate diseases.”

http://www.ncbi.nlm.nih.gov/pmc/articles/PMC3464034/

Cannabis: Time for Scientific Evaluation of This Ancient Remedy?

” It is clear, therefore, that there is sufficient anecdotal clinical data and sound experimental data to encourage proper scientific evaluation of the potential therapeutic benefit of cannabis. Although it undoubtedly possesses the potential for psychic and physical dependency, these are considerably less than the undesirable effects of opioids. The time is now appropriate, therefore, for proper evaluation of this ancient remedy.”

http://www.anesthesia-analgesia.org/content/90/2/237.long

Cannabinoid receptor 1 is a potential drug target for treatment of translocation-positive rhabdomyosarcoma

“Because cannabinoid receptor agonists are capable of reducing proliferation and inducing apoptosis (cell death) in diverse cancer cells such as glioma, breast cancer, and melanoma, we evaluated whether CB1 is a potential drug target in rhabdomyosarcoma.

Rhabdomyosarcoma (RMS) is the most common soft-tissue sarcoma in children…

 Our study shows that treatment with the cannabinoid receptor agonists HU210 (cloned THC from Hebrew University) and Delta(9)-tetrahydrocannabinol (THC from cannabis) lowers the viability of translocation-positive rhabdomyosarcoma cells through the induction of apoptosis…

These results support the notion that cannabinoid receptor agonists could represent a novel targeted approach for treatment of translocation-positive rhabdomyosarcoma.”

Full text: http://mct.aacrjournals.org/content/8/7/1838.long

The CB1 Cannabinoid Receptor Is Coupled to the Activation of c-Jun N-Terminal Kinase

“One of the most intriguing and unexplored actions of cannabinoids is their ability to control cell growth. Thus, cannabinoids have been shown to induce antiproliferative effects through the CB1 receptor… we have recently shown that cannabinoids are able to modulate through the CB1 receptor the activity of the PI3K/protein kinase B pathway, which serves as a pivotal antiapoptotic signal…”

http://molpharm.aspetjournals.org/content/58/4/814.long

Cannabidiol-induced apoptosis in primary lymphocytes is associated with oxidative stress-dependent activation of caspase-8

“Cannabidiol-induced apoptosis in primary lymphocytes is associated with oxidative stress-dependent activation of caspase-8” and “The non-psychoactive cannabidiol triggers caspase activation and oxidative stress in human glioma cells… we have shown that the non-psychoactive cannabinoid compound cannabidiol (CBD) induces apoptosis (cell death) of glioma cells in vitro and tumor regression…”

http://www.ncbi.nlm.nih.gov/pubmed/17950393

Cannabinoids: A new hope for breast cancer therapy?

“Experimental evidence accumulated during the last decade supports that cannabinoids, the active components of Cannabis sativa and their derivatives, possess anticancer activity… these compounds exert anti-proliferative, pro-apoptotic, anti-migratory and anti-invasive actions in a wide spectrum of cancer cells… Moreover, tumor growth, angiogenesis and metastasis are hampered by cannabinoids… our current knowledge on the anti-tumor potential of cannabinoids in breast cancer… suggests that cannabinoid-based medicines may be useful for the treatment of most breast tumor subtypes.”

http://www.ncbi.nlm.nih.gov/pubmed/22776349 

Rimonabant inhibits human colon cancer cell growth and reduces the formation of precancerous lesions in the mouse colon

“Rimonabant inhibits human colon cancer cell growth and reduces the formation of precancerous lesions…” and “Cannabinoid receptor activation induces apoptosis… in colon cancer cells”

http://www.ncbi.nlm.nih.gov/pubmed/19479993

Cannabis and tobacco smoke are not equally carcinogenic

“Cannabis and tobacco smoke are not equally carcinogenic… Cannabis smoke contains cannabinoids whereas tobacco smoke contains nicotine (plus added carcinogens). Available scientific data, that examines the carcinogenic properties of inhaling smoke and its biological consequences, suggests reasons why tobacco smoke, but not cannabis smoke, may result in lung cancer… Furthermore, compounds found in cannabis have been shown to kill numerous cancer types including: lung cancer, breast and prostate, leukemia and lymphoma, glioma, skin cancer, and pheochromocytoma…”-

http://www.ncbi.nlm.nih.gov/pmc/articles/PMC1277837/?tool=pubmed