It’s Lung Cancer Awareness Month. Please, BE AWARE:

A tourist looks over the city's skyline over a thin haze of pollution on a clear day in Hong Kong on February 1, 2013. Emissions from factories in southern China, which seep over Hong Kong's border, combined with local emissions from power plants and transport, generate an almost daily thick blanket of haze over the teeming metropolis.

“Air pollution causes lung cancer, World Health Organization confirms” http://www.cbsnews.com/8301-204_162-57607955/air-pollution-causes-lung-cancer-world-health-organization-confirms/

“Marijuana May Fight Lung Tumors… active ingredient in marijuana may help combat lung cancer… In lab and mouse studies, the compound, known as THC, cut lung tumor growth in half and helped prevent the cancer from spreading…” http://www.cbsnews.com/2100-500368_162-2696726.html

“Lung cancer leads all cancer deaths”  http://www.thespectrum.com/story/life/features/mesquite/2014/10/20/lung-cancer-leads-cancer-deaths/17640075/

“Air Pollution a Leading Cause of Cancer. The United Nation’s World Health Organization (WHO) has classified air pollution as a prime cause of cancer worldwide, especially in the case of lung cancer.” http://www.webmd.com/cancer/news/20131018/air-pollution-cancer

“Pot Smoking Not Linked to Lung Cancer. Study Shows No Increased Risk for Even the Heaviest Marijuana Smokers.” http://www.webmd.com/lung-cancer/news/20060523/pot-smoking-not-linked-to-lung-cancer

“Marijuana May Fight Lung Tumors… the active ingredient in marijuana may help combat lung cancer…”http://www.webmd.com/lung-cancer/news/20070417/marijuana-may-fight-lung-tumors

“WHO agency: Air pollution causes cancer. What many commuters choking on smog have long suspected has finally been scientifically validated: air pollution causes lung cancer.” http://www.abclocal.go.com/ktrk/story?section=news%2Fhealth&id=9290406

“Marijuana Compound May Fight Lung Cancer… Harvard University researchers have found that, in both laboratory and mouse studies, delta-tetrahydrocannabinol (THC) cuts tumor growth in half in common lung cancer while impeding the cancer’s ability to spread.”
http://abcnews.go.com/Health/Healthday/story?id=4506595&page=1

“Don’t Smoke? You Could Still Get Lung Cancer”  http://health.clevelandclinic.org/2014/10/dont-smoke-you-could-still-get-lung-cancer/

“Radon No. 1 lung cancer cause in nonsmokers”  http://www.rgj.com/story/life/wellness/2014/10/24/radon-lung-cancer-cause-nonsmokers/17866569/

“Air pollution causes lung cancer, World Health Organization says”  http://articles.latimes.com/2013/oct/17/science/la-sci-sn-air-pollution-causes-lung-cancer-20131017

“Smog Tied to Higher Risk of Lung Cancer, Heart Failure” http://health.usnews.com/health-news/news/articles/2013/07/10/smog-tied-to-higher-risk-of-lung-cancer-heart-failure

“Mountaintop Removal Mining Dust Linked To Increased Risk Of Lung Cancer, According to Study”  http://lungcancernewstoday.com/2014/10/20/mountaintop-removal-mining-dust-linked-increased-risk-lung-cancer-according-study/

“Study Shows Direct Connections Between Mountaintop Removal Dust Exposure and Lung Cancer”  http://wfpl.org/post/study-shows-direct-connections-between-mountaintop-removal-dust-exposure-and-lung-cancer

“Compound in chili pepper slows lung cancer tumor growth. New laboratory research shows capsaicin decrease growth of an aggressive form of lung cancer.”  http://www.teatronaturale.com/food-and-fun/health/4983-compound-in-chili-pepper-slows-lung-cancer-tumor-growth.htm

“Drinking alcohol may boost lung cancer risk” http://www.foodconsumer.org/newsite/Nutrition/Food/drinking_alcohol_and_lung_cancer_risk_1024110625.html

“Link Between Alcohol And Cancer Explained: Alcohol Activates Cellular Changes That Make Tumor Cells Spread” http://www.sciencedaily.com/releases/2009/10/091026172052.htm

“Groups seek to curb tobacco use in cancer patients… Tobacco use plays a role in 18 different cancers… Patients who use tobacco have worse outcomes and more difficult treatment… tobacco use decreases the effectiveness of chemotherapies and interferes with drug metabolism… data also shows that it decreases survival… we call on the oncology community… to provide cessation support to all cancer patients,” http://www.oncologypractice.com/oncologyreport/news/top-news/single-view/groups-seek-to-curb-tobacco-use-in-cancer-patients/1a87bf57bcccdadf01ae769c48eadce4.html

“Marijuana habit not linked to lung cancer” http://www.oncologypractice.com/oncologyreport/news/top-news/single-view/marijuana-habit-not-linked-to-lung-cancer/73840afd2cca226b9e6a9ddc7cb0d039.html

“Cannabis and tobacco smoke are not equally carcinogenic… Available scientific data, that examines the carcinogenic properties of inhaling smoke and its biological consequences, suggests reasons why tobacco smoke, but not cannabis smoke, may result in lung cancer… Furthermore, compounds found in cannabis have been shown to kill numerous cancer types including: lung cancer…”  http://www.harmreductionjournal.com/content/2/1/21

“Tobacco Smoke Causes Lung Inflammation, Promotes Lung Cancer Growth” http://www.sciencedaily.com/releases/2010/01/100119121206.htm

“Marijuana Cuts Lung Cancer Tumor Growth In Half, Study Shows”
http://www.sciencedaily.com/releases/2007/04/070417193338.htm

“Delta–9 Tetrahydrocannabinol inhibits growth and metastasis of lung cancer” http://www.thctotalhealthcare.com/delta-9-tetrahydrocannabinol-inhibits-growth-and-metastasis-of-lung-cancer-harvard-university/

“Δ-9 Tetrahydrocannabinol inhibits growth and metastasis of lung cancer.”  http://cancerres.aacrjournals.org/content/67/9_Supplement/4749.short

“Antineoplastic activity of cannabinoids. Lewis lung adenocarcinoma growth was retarded by the oral administration of delta-9-tetrahydrocannabinol, delta-8-tetrahydrocannabinol, and cannabinol (CBN)… Delta-9-THC, delta-8-THC, and cannabinol (CBN) all inhibited primary Lewis lung tumor growth….. All occur naturally in marihuana… these compounds readily cross the blood-brain barrier and do not possess many of the toxic manifestations of presently used cytotoxic agents…” http://www.ukcia.org/research/AntineoplasticActivityOfCannabinoids/index.php

“Antineoplastic activity of cannabinoids.”  http://www.ncbi.nlm.nih.gov/pubmed/1159836

“Antineoplastic: A drug used to inhibit the growth and spread of cancerous cells.. a chemotherapeutic agent that controls or kills cancer cells…” http://medical-dictionary.thefreedictionary.com/antineoplastic

“The Inhibition of DNA Synthesis by Cannabinoids. Several of the cannabinoids found in marihuana have been shown to inhibit tumor growth and increase the life-span… our in vitro observations with these cannabinoids are supported by in vivo tumor inhibition studies… Δ9-trans-tetrahydrocannabinol demonstrated the least toxicity of all inhibitory cannabinoids in vivo…” http://cancerres.aacrjournals.org/content/36/1/95.short

“Inhibition: “arrest or restraint of a process… Something that restrains, blocks, or suppresses… (in chemistry) the stopping or slowing of the rate of a chemical reaction.” http://medical-dictionary.thefreedictionary.com/inhibition

“Effects of delta9-tetrahydrocannabinol in Lewis lung adenocarcinoma cells in tissue culture… We found a dose-related decrease in DNA synthesis in transformed cell cultures treated with delta9-tetrahydrocannabinol (delta9-THC)… delta9-THC inhibited DNA synthesis…” http://www.ncbi.nlm.nih.gov/pubmed/943561

“In vivo effects of cannabinoids on macromolecular biosynthesis in Lewis lung carcinomas. Cannabinoids represent a novel class of drugs active in increasing the life span mice carrying Lewis lung tumors and decreasing primary tumor size…” http://www.ncbi.nlm.nih.gov/pubmed/616322

“Anti-emetic efficacy and toxicity of nabilone, a synthetic cannabinoid, in lung cancer chemotherapy… Symptom scores were significantly better for patients on nabilone for nausea, retching and vomiting. Fewer subjects vomited with nabilone and the number of vomiting episodes was lower; no patients on nabilone required additional parenteral anti-emetic. More patients preferred nabilone for anti-emetic control… Nabilone is an effective oral anti-emetic drug for moderately toxic chemotherapy, but the range and unpredictability of its side-effects warrant caution in its use.” http://www.ncbi.nlm.nih.gov/pmc/articles/PMC2011510/

Antiemetic: “preventing or alleviating nausea and vomiting…” http://medical-dictionary.thefreedictionary.com/antiemetic
“Double-blind comparison of the antiemetic effects of nabilone and prochlorperazine on chemotherapy-induced emesis. The antiemetic effect of oral nabilone, a synthetic cannabinoid, was compared to oral slow-release capsules of prochlorperazine… Nabilone appeared to be the more effective antiemetic…” http://www.ncbi.nlm.nih.gov/pubmed/6250699
“Superiority of nabilone over prochlorperazine as an antiemetic in patients receiving cancer chemotherapy. Two double-blind, crossover trials comparing the antiemetic effectiveness of nabilone, a new synthetic cannabinoid, with that of prochlorperazine were conducted in patients with severe nausea and vomiting associated with anticancer chemotherapy… patients clearly favored nabilone…” http://www.ncbi.nlm.nih.gov/pubmed/375088
“Nabilone versus prochlorperazine for control of cancer chemotherapy-induced emesis in children: a double-blind, crossover trial… Nabilone appears to be a safe, effective, and well-tolerated antiemetic drug for children receiving cancer chemotherapy. Although major side effects may occur at higher dosages, nabilone is preferable to prochlorperazine because of improved efficacy.” http://www.ncbi.nlm.nih.gov/pubmed/3035479
“Nabilone: an effective antiemetic in patients receiving cancer chemotherapy… Eighty evaluable patients receiving chemotherapy were entered on a random prospective double-blind study to evaluate the effecti…veness of nabilone, a synthetic cannabinoid, compared to prochlorperazine… Sixty patients (75 per cent) reported nabilone to be more effective than prochlorperazine for relief of nausea and vomiting.” http://www.ncbi.nlm.nih.gov/pubmed/6271844
“Antiemetic effect of delta-9-tetrahydrocannabinol in patients receiving cancer chemotherapy. Anecdotal accounts suggested that smoking marihuana decreases the nausea and vomiting associated with cancer chemotherapeutic agents. Oral delta-9-tetrahydrocannabinol was compared with placebo in a controlled, randomized, “double-blind” experiment… For all patients an antiemetic effect was observed… No patient vomited while experiencing a subjective “high”. Oral tetrahydrocannabinol has antiemetic properties and is significantly better than a placebo in reducting vomiting caused by chemotherapeutic agents.” http://www.ncbi.nlm.nih.gov/pubmed/1099449
“… a randomized, double-blind, placebo-controlled trial of oral and smoked delta-9-tetrahydrocannabinol (THC) as an antiemetic… Delta-9-tetrahydrocannabinol was significantly more effective than placebo in reducing the number of vomiting and retching episodes, degree of nausea, duration of nausea, and volume of emesis… Delta-9-tetrahydrocannabinol appears to have significant antiemetic properties…” http://www.ncbi.nlm.nih.gov/pubmed/293141
“Antiemetic effect of tetrahydrocannabinol. Compared with placebo and prochlorperazine in chemotherapy-associated nausea and emesis… The antiemetic effect of THC appeared to be more efficacious… Tetrahydrocannabinol appears to offer significant control of nausea in most patients and exceeding by far that provided by prochlorperazine.”
http://www.ncbi.nlm.nih.gov/pubmed/6254456
“Antiemetic effect of delta 9-tetrahydrocannabinol in chemotherapy-associated nausea and emesis as compared to placebo and compazine… THC appeared to be more efficacious in controlling the emesis… THC appears to offer significant control of nausea in most patients…” http://www.ncbi.nlm.nih.gov/pubmed/6271846
“Delta-9-tetrahydrocannabinol as an antiemetic… THC appears to be an effective antiemetic in cancer patients undergoing chemotherapy… THC has been found consistently more effective than placebo and at least as effective as prochlorperazine… THC’s effectiveness has exceeded that of the phenothiazines.” http://www.ncbi.nlm.nih.gov/pubmed/6269423
“Efficacy of tetrahydrocannabinol in patients refractory to standard antiemetic therapy… This study confirms the usefulness of THC in patients whose chemotherapy-induced nausea and vomiting is refractory to other standard antiemetics.” http://www.ncbi.nlm.nih.gov/pubmed/2847994

“Cannabidiol inhibits lung cancer cell invasion and metastasis via intercellular adhesion molecule-1.”  http://www.ncbi.nlm.nih.gov/pubmed/22198381

“Cannabidiol inhibits cancer cell invasion via upregulation of tissue inhibitor of matrix metalloproteinases-1.” http://www.ncbi.nlm.nih.gov/pubmed/19914218

“Decrease of plasminogen activator inhibitor-1 may contribute to the anti-invasive action of cannabidiol on human lung cancer cells” http://www.ncbi.nlm.nih.gov/pubmed/20668920

“CYCLOOXYGENASE-2 AND PEROXISOME PROLIFERATOR ACTIVATED RECEPTOR γ CONFER CANNABIDIOL-INDUCED APOPTOSIS OF HUMAN LUNG CANCER CELLS.” http://www.ncbi.nlm.nih.gov/pubmed/23220503 

“Apoptosis: programmed cell death at a molecular level.” http://www.ncbi.nlm.nih.gov/pubmed/12833244

“WHO agency says air pollution causes lung cancer” http://www.foxnews.com/health/2013/10/17/who-agency-says-air-pollution-causes-lung-cancer/

“Marijuana Does Not Raise Lung Cancer Risk” http://www.foxnews.com/story/0,2933,196678,00.html

“Marijuana May Fight Lung Tumors… marijuana may help combat lung cancer… In lab and mouse studies, the compound, known as THC, cut lung tumor growth in half and helped prevent the cancer from spreading, says Anju Preet, PhD, a Harvard University researcher in Boston…”http://www.foxnews.com/story/2007/04/18/marijuana-may-fight-lung-tumors/

“Air pollution causes lung cancer, WHO agency announces. Experts at the International Agency for Research on Cancer, an agency of the World Health Organization, have concluded that exposure to outdoor air pollution.causes lung cancer and increases the risk for bladder cancer.” http://www.nbcnews.com/video/nightly-news/53309399/#53309399

“Marijuana Compound Fights Cancer; Human Trials Next.
Research shows component in medical cannabis fights cancer.” http://www.nbcbayarea.com/news/local/Marijuana-Compound-Fights-Cancer-Human-Trials-Next-170406116.html

“The endocannabinoid system and cancer: therapeutic implication… Identification of safe and effective treatments to manage and improve cancer therapy is critical to improve quality of life and reduce unnecessary suffering in cancer patients. In this regard, cannabis-like compounds offer therapeutic potential for the treatment of… cancer… anti-cancer properties of cannabinoids…” http://www.ncbi.nlm.nih.gov/pmc/articles/PMC3165955/

“The endocannabinoid system in the cancer therapy: an overview.” http://www.ncbi.nlm.nih.gov/pubmed/21428888

“The endocannabinoid signaling system in cancer.”  http://www.ncbi.nlm.nih.gov/pubmed/23602129

“Changes in the Endocannabinoid System May Give Insight into new and Effective Treatments for Cancer” http://www.ncbi.nlm.nih.gov/pmc/articles/PMC2791688/

“Endocannabinoid system modulation in cancer biology and therapy.” http://www.ncbi.nlm.nih.gov/pubmed/19559362

“The endocannabinoid system in cancer-potential therapeutic target?” http://www.ncbi.nlm.nih.gov/pubmed/18249558

“Targeting the endocannabinoid system for the treatment of cancer–a practical view.” http://www.ncbi.nlm.nih.gov/pubmed/20370711

“The endocannabinoid system and its therapeutic exploitation… since the discovery of cannabinoid receptors, the endocannabinoid system seems to hold even more promise for the future development of therapeutic drugs.” http://www.ncbi.nlm.nih.gov/pubmed/15340387

“The endocannabinoid system as a target for the development of new drugs for cancer therapy… evidence has accumulated indicating that stimulation of cannabinoid receptors by either THC or the endocannabinoids influence the intracellular events controlling the proliferation and apoptosis of numerous types of cancer cells, thereby leading to anti-tumour effects both in vitro and in vivo. This evidence is reviewed here and suggests that future anti-cancer therapy might be developed from our knowledge of how the endocannabinoid system controls the growth and metastasis of malignant cells.”
http://www.ncbi.nlm.nih.gov/pubmed/12723496

“Cannabinoid receptor systems: therapeutic targets for tumour intervention… there is now a genuine expectation that the manipulation of cannabinoid receptor systems may have therapeutic potential for a diverse range of human diseases… demonstrated antitumour actions of cannabinoids indicates possible avenues for the future development of cannabinoids as antitumour agents.”
http://www.ncbi.nlm.nih.gov/pubmed/14640910

“Cannabinoid-associated cell death mechanisms in tumor models… cannabinoids (the active components of Cannabis sativa)… in addition to their inhibitory effects on tumor growth and migration, angiogenesis and metastasis… these compounds induce different pathways of cell death… death mechanisms induced by cannabinoids… we analyze the pathways triggered by cannabinoids to induce apoptosis or autophagy… the results reported here suggest that the exploration of molecular mechanisms induced by cannabinoids in cancer cells can contribute to the development of safe and effective treatments in cancer therapy.” http://www.ncbi.nlm.nih.gov/pubmed/22614735

“Towards the use of cannabinoids as antitumour agents.. cannabinoids (the active components of marijuana and their derivatives) can reduce tumour growth and progression in animal models of cancer, in addition to their well-known palliative effects on some cancer-associated symptoms.”
http://www.ncbi.nlm.nih.gov/pubmed/22555283

“Non-small cell lung cancer (NSCLC) is the leading cause of cancer deaths worldwide…only limited therapeutic treatments are available. Hence, we investigated the role of cannabinoid receptors, CB1 and CB2, as novel therapeutic targets against NSCLC…. Overall, our results indicate a novel role for cannabinoid receptors CB1 and CB2 in NSCLC growth and metastasis… These results suggest that CB1 and CB2 could be used as novel therapeutic targets against NSCLC.”
http://www.ncbi.nlm.nih.gov/pmc/articles/PMC3025486/

“Cannabinoid receptors as therapeutic targets.”
http://www.ncbi.nlm.nih.gov/pubmed/16402900

“Inhibitory effects of cannabinoid CB1 receptor stimulation on tumor growth and metastatic spreading: actions on signals involved in angiogenesis and metastasis… Our findings indicate that CB1 receptor agonists might be used therapeutically to retard tumor growth in vivo by inhibiting at once tumor growth, angiogenesis, and metastasis.” http://www.ncbi.nlm.nih.gov/pubmed/12958205

“Targeting the endocannabinoid system with cannabinoid receptor agonists: pharmacological strategies and therapeutic possibilities. Human tissues express cannabinoid CB(1) and CB(2) receptors that can be activated by endogenously released ‘endocannabinoids’ or exogenously administered compounds in a manner that reduces the symptoms or opposes the underlying causes of several disorders in need of effective therapy. Three medicines that activate cannabinoid CB(1)/CB(2) receptors are now in the clinic: Cesamet (nabilone), Marinol (dronabinol; Δ(9)-tetrahydrocannabinol (Δ(9)-THC)) and Sativex (Δ(9)-THC with cannabidiol)… several possible additional therapeutic targets for cannabinoid receptor agonists. These include other kinds of pain, epilepsy, anxiety, depression, Parkinson’s and Huntington’s diseases, amyotrophic lateral sclerosis, stroke, CANCER, drug dependence, glaucoma, autoimmune uveitis, osteoporosis, sepsis, and hepatic, renal, intestinal and cardiovascular disorders.”
http://www.ncbi.nlm.nih.gov/pubmed/23108552

“Cannabinoid receptor agonists are mitochondrial inhibitors: a unified hypothesis of how cannabinoids modulate mitochondrial function and induce cell death. Time-lapse microscopy of human lung cancer cells showed t…hat the endogenous cannabinoid anandamide (AEA), the phyto-cannabinoid Delta-9-tetrahydrocannabinol (THC) and a synthetic cannabinoid HU 210 all caused morphological changes characteristic of apoptosis.” http://www.ncbi.nlm.nih.gov/pubmed/17931597

“Programmed Cell Death (Apoptosis)” http://www.ncbi.nlm.nih.gov/books/NBK26873/

“Δ9-Tetrahydrocannabinol inhibits epithelial growth factor-induced lung cancer cell migration in vitro as well as its growth and metastasis in vivo…Tumor samples from THC-treated animals revealed antiproliferative and antiangiogenic effects of THC. Our study suggests that cannabinoids like THC should be explored as novel therapeutic molecules in controlling the growth and metastasis of certain lung cancers.” http://www.nature.com/onc/journal/v27/n3/abs/1210641a.html

“Use of cannabinoid receptor agonists in cancer therapy as palliative and curative agents. Cannabinoids (the active components of Cannabis sativa)… evidence suggests that agonists of cannabinoid receptors expressed by tumour cells may offer a novel strategy to treat cancer… use of cannabinoid agonists for cancer therapy, not only as palliative but also as curative drugs.” http://www.ncbi.nlm.nih.gov/pubmed/19285265

http://www.thctotalhealthcare.com/category/lung-cancer/

Pot Slows Cancer in Test Tube – WebMD

“THC and another marijuana-derived compound slow the spread of cervical and lung cancers, test-tube studies suggest.

The new findings add to the fast-growing number of animal and cell-culture studies showing different anticancer effects for cannabinoids, chemical compounds derived from marijuana.

Cannabinoids, and sometimes marijuana itself, are currently used to lessen the nausea and pain experienced by many cancer patients. The new findings — suggest that cannabinoids may have a direct anticancer effect.

“Cannabinoids’ … potential therapeutic benefit in the treatment of highly invasive cancers should be addressed in clinical trials,” conclude Robert Ramer, PhD, and Burkhard Hinz, PhD, of the University of Rostock, Germany…

Doses of THC that reduce pain in cancer patients yield blood concentrations much higher than the concentrations needed to inhibit cancer invasion.

“Thus the effects of THC on cell invasion occurred at therapeutically relevant concentrations,” Ramer and Hinz note…

Ramer and Hinz report the findings in the Jan. 2, 2008 issue of the Journal of the National Cancer Institute.”

More: http://www.webmd.com/cancer/news/20071226/pot-slows-cancer-in-test-tube

“Inhibition of Cancer Cell Invasion by Cannabinoids via Increased Expression of Tissue Inhibitor of Matrix Metalloproteinases-1”: http://jnci.oxfordjournals.org/content/100/1/59.long

Marijuana Inhibits Tumors

Therapeutic potential of cannabinoid medicines.

Drug Testing and Analysis

“Cannabis was extensively used as a medicine throughout the developed world in the nineteenth century but went into decline early in the twentieth century ahead of its emergence as the most widely used illicit recreational drug later that century. Recent advances in cannabinoid pharmacology alongside the discovery of the endocannabinoid system (ECS) have re-ignited interest in cannabis-based medicines.

The ECS has emerged as an important physiological system and plausible target for new medicines. Its receptors and endogenous ligands play a vital modulatory role in diverse functions including immune response, food intake, cognition, emotion, perception, behavioural reinforcement, motor co-ordination, body temperature, wake/sleep cycle, bone formation and resorption, and various aspects of hormonal control. In disease it may act as part of the physiological response or as a component of the underlying pathology.

In the forefront of clinical research are the cannabinoids delta-9-tetrahydrocannabinol and cannabidiol, and their contrasting pharmacology will be briefly outlined. The therapeutic potential and possible risks of drugs that inhibit the ECS will also be considered. This paper will then go on to review clinical research exploring the potential of cannabinoid medicines in the following indications: symptomatic relief in multiple sclerosis, chronic neuropathic pain, intractable nausea and vomiting, loss of appetite and weight in the context of cancer or AIDS, psychosis, epilepsy, addiction, and metabolic disorders.”

http://www.ncbi.nlm.nih.gov/pubmed/24006213

http://onlinelibrary.wiley.com/doi/10.1002/dta.1529/abstract

Smokin’: Report finds marijuana has negligible effects on lungs

“According to a new study, habitual use of marijuana alone doesn’t appear to lead to significant abnormalities in lung function, nor does it increase the risks of COPD or either lung or upper airway cancer.”

“The pulmonary consequences of regularly smoking marijuana are far less than for tobacco, according a review of the published evidence conducted by Dr. Donald P. Tashkin, emeritus professor of medicine and medical director of the pulmonary function laboratory at the University of California, Los Angeles. “

More: http://www.familypracticenews.com/single-view/smokin-report-finds-marijuana-has-negligible-effects-on-lungs/e06976673e7c44157d83649773e81193.html

 

 

Study Finds No Link Between Marijuana Use And Lung Cancer – ScienceDaily

“People who smoke marijuana–even heavy, long-term marijuana users–do not appear to be at increased risk of developing lung cancer…

Marijuana smoking also did not appear to increase the risk of head and neck cancers, such as cancer of the tongue, mouth, throat, or esophagus, the study found.

The findings were a surprise to the researchers. “We expected that we would find that a history of heavy marijuana use–more than 500-1,000 uses–would increase the risk of cancer from several years to decades after exposure to marijuana,” said the senior researcher, Donald Tashkin, M.D., Professor of Medicine at the David Geffen School of Medicine at UCLA in Los Angeles.”

More: http://www.sciencedaily.com/releases/2006/05/060526083353.htm

Large Study Finds No Link between Marijuana and Lung Cancer – Scientific American

“…Scientists were therefore surprised to learn that a study of more than 2,000 people found no increase in the risk of developing lung cancer for marijuana smokers.

“We expected that we would find that a history of heavy marijuana use–more than 500 to 1,000 uses–would increase the risk of cancer from several years to decades after exposure to marijuana,” explains physician Donald Tashkin of the University of California, Los Angeles, and lead researcher on the project. But looking at residents of Los Angeles County, the scientists found that even those who smoked more than 20,000 joints in their life did not have an increased risk of lung cancer.

The study does not reveal how marijuana avoids causing cancer. Tashkin speculates that perhaps the THC chemical in marijuana smoke prompts aging cells to die before becoming cancerous. Tashkin and his colleagues presented the findings yesterday at a meeting of the American Thoracic Society in San Diego.”

More: http://www.scientificamerican.com/article.cfm?id=large-study-finds-no-link

Marijuana Smoking Not Linked To Cancer or Lung Damage, Researchers Say

 “Donald Tashkin’s is a tale cannabis pushers like to repeat. The physician and professor at UCLA’s David Geffen School of Medicine set out to prove — via a study funded by the National Institutes on Drug Abuse — that marijuana is bad for you. Instead, a long-term study found no solid link between marijuana use and lung cancer, in sharp contrast to tobacco terrible effects on health.” 

donald-tashkin.jpg

“Similar findings were repeated all over the world. In a collection and review of studies on marijuana’s effect on the lungs, published in the June issue of the Annals of the American Thoracic Society, Tashkin concludes that compared to tobacco smoking, heavy marijuana use has “relatively small and far lower” risks.

This despite an average joint marijuana having four times the tar of a typical American Spirit. How can this be?

It’s worth remembering that this is not a new development — Tashkin’s long-term study was published in 2006. And well before that — as in the 19th Century, when cannabis tinctures and other marijuana medicines were sold in pharmacies — doctors were prescribing marijuana as a treatment for asthma patients.”

More: http://blogs.sfweekly.com/thesnitch/2013/06/marijuana_cancer_annals_of_the_american_thoracic_society.php

Breathe Easy: A Marijuana Study Finds No Lung Cancer Links

Donald Tashkin‘s is a tale cannabis pushers like to repeat. The physician and professor at UCLA‘s David Geffen School of Medicine set out to prove — via a study funded by the National Institutes on Drug Abuse — that marijuana is bad for you. Instead, a long-term study found no solid link between marijuana use and lung cancer.

Similar findings were repeated all over the world. In a review of studies on marijuana’s effect on the lungs, published in the June issue of the Annals of the American Thoracic Society, Tashkin concludes that compared to tobacco smoking, heavy marijuana use has “relatively small and far lower” risks.”

More: http://www.sfweekly.com/2013-06-19/news/ucla-medical-marijuana-cancer/full/

Cannabis Ingredient Can Help Cancer Patients Regain Their Appetites And Sense Of Taste

MNT home

“The active ingredient in cannabis can improve the appetites and sense of taste in cancer patients, according to a new study published online in the cancer journal, Annals of Oncology  today.

Loss of appetite is common among cancer patients, either because the cancer itself or its treatment affects the sense of taste and smell, leading to decreased enjoyment of food. This, in turn, can lead to weight loss, anorexia, a worse quality of life and decreased survival; therefore, finding effective ways of helping patients to maintain a good diet and consume enough calories is an important aspect of their treatment.

The majority of THC-treated patients (64%) had increased appetite, three patients (27%) showed no change, and one patient’s data was incomplete. No THC-treated patients showed a decrease in appetite. By contrast, the majority of patients receiving placebo had either decreased appetite (50%) or showed no change (20%).

Although there was no difference in the total number of calories consumed by both groups, the THC-treated patients tended to increase the proportion of protein that they ate, and 55% reported that savoury foods tasted better, whereas no patients in the placebo group reported an increased liking for these foods. (Cancer patients often find that meat smells and tastes unpleasant and, therefore, they eat less of it).

In addition, THC-treated patients reported better quality of sleep and relaxation than in the placebo group.”

More:  http://www.medicalnewstoday.com/articles/217062.php